在摄入聚氨泡后胃孔穿孔
Ki Bum Park1, Woo Young Nho2, Byunghyuk Yu3
1Department of Surgery, School of Medicine, Kyungpook National University, Kyungpook National University Chilgok Hospital, Daegu, Republic of Korea.
Clinical toxicology (Philadelphia, Pa.)
|April 8, 2024
概括
摄入聚氨泡可能会导致危及生命的胃穿孔. 医疗专业人员应该认识到这种有毒物质的可扩展性.
科学领域:
- 毒理学 毒理学 毒理学
- 胃肠病学 胃肠病学
- 紧急医疗 紧急医疗
背景情况:
- 急性中毒很少需要手术干预.
- 摄入聚氨是一种不常见的手术紧急情况的原因.
研究的目的:
- 报告一种罕见的胃穿孔病例,由于摄入聚氨.
- 强调认识到聚氨在毒理紧急情况中的可扩展性的重要性.
主要方法:
- 一名45岁的男性在摄入聚氨泡后出现了严重的腹痛.
- 诊断性腹腔镜检查发现了异物物质和血液皮质,导致紧急探索性腹腔切除术.
- 进行了从胃和食道中切除固化聚氨件的手术.
主要成果:
- 计算机断层扫描证实胃腔内有大量的外来物质和扩散的胃穿孔.
- 手术探索发现并成功地去除了固化聚氨件,包括胃和食道的模具.
- 病人康复了,手术后14天就出院了.
结论:
- 危及生命的胃穿孔是摄入聚氨泡的潜在并发症.
- 医学毒理学家和急诊医生必须意识到摄入聚氨的显著扩张潜力.
- 迅速的手术干预对于管理摄入聚氨造成的严重胃肠道并发症至关重要.
相关概念视频
Esophageal Perforation-I: Introduction
91
Esophageal perforation is a severe medical condition characterized by a breach in the integrity of the esophageal wall. This breach can occur due to various factors such as trauma, medical procedures, or underlying diseases. When the esophageal wall is compromised, it allows food, fluids, and digestive juices into the chest cavity or adjacent structures, leading to potential complications and health risks.
The location of esophageal perforation can vary, occurring anywhere along the esophagus....
The location of esophageal perforation can vary, occurring anywhere along the esophagus....
91
Esophageal Perforation-II: Clinical Manifestations and Management
58
Esophageal perforations manifest in various clinical forms, influenced by factors such as the perforation's cause and location (cervical, intrathoracic, or intra-abdominal), the extent of contamination, and potential injury to adjacent mediastinal structures. The timing between the perforation occurrence and treatment initiation also affects the clinical presentation.
Clinical Manifestations:
Clinical Manifestations:
58
Peptic Ulcer Disease I: Introduction
166
Peptic Ulcer Disease (PUD) is characterized by mucosal excavation in the esophagus, stomach, pylorus, or duodenum. It can manifest as acute or chronic based on the extent and duration of mucosal involvement.
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
166
Peptic Ulcer Disease V: Surgical Management and Nursing Care
299
Surgical management and nursing care are crucial in treating Peptic Ulcer Disease (PUD). Here is an organized and enhanced overview of the surgical interventions and the associated nursing care for PUD:
Surgical Interventions for Peptic Ulcer Disease
Surgical Interventions for Peptic Ulcer Disease
299
Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies
116
Peptic ulcer disease (PUD) presents with diverse symptoms depending on the location and severity of the ulcer. Clinical manifestations of peptic ulcer include dull pain and a burning sensation in the mid-epigastric region.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
116
Peptic Ulcer Disease II: Pathophysiology
412
Peptic Ulcer Disease (PUD) is characterized by the development of ulcers in the stomach or duodenal mucosa. Its pathophysiology is complex, involving a balance between damaging and protective elements.
Damaging agents such as Helicobacter pylori, gastric acid, pepsin, and nonsteroidal anti-inflammatory drugs (NSAIDs) can weaken the mucosal defense, allowing hydrogen ions to infiltrate back and harm epithelial cells.
Damaging agents such as Helicobacter pylori, gastric acid, pepsin, and nonsteroidal anti-inflammatory drugs (NSAIDs) can weaken the mucosal defense, allowing hydrogen ions to infiltrate back and harm epithelial cells.
412


