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Updated: Jun 29, 2025

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蛋白酶3的枯竭通过促进髓状细胞分化来减轻白血病

Huan Liu1,2, Lu Sun1,2, Hongfei Zhao1,2

  • 1State Key Laboratory of Experimental Hematology, National Clinical Research Center for Blood Diseases, Haihe Laboratory of Cell Ecosystem, Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Sciences & Peking Union Medical College, Tianjin, 300020, China.

Cell death and differentiation
|April 8, 2024
PubMed
概括

蛋白酶3 (PRTN3) 的遗传消除通过调节STAT3.3促进骨髓分化. 在急性髓性白血病 (AML) 细胞中,PRTN3 缺乏会增强它们的分化,提高生存率.

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Common myeloid progenitors (CMPs) are oligopotent cells that can differentiate into granulocytes and macrophages. Granulocytes and macrophages are essential for protecting the body against bacterial, viral, or fungal infections. They migrate from the bone marrow into the circulating blood to reach specific tissue sites where they differentiate and help in immune surveillance. However, they survive only for a few days and must be continuously made available to the organism to maintain a robust...
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