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Updated: Jun 28, 2025

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在急性髓性白血病中B7-H3:从预后生物标志物到免疫治疗点

Xiao Tan1, Xiangyu Zhao

  • 1Peking University People's Hospital, Peking University Institute of Hematology, National Clinical Research Center for Hematologic Disease, Beijing Key Laboratory of Hematopoietic Stem Cell Transplantation, Beijing 100044, China.

Chinese medical journal
|April 10, 2024
PubMed
概括

B7-H3在急性髓性白血病 (AML) 中过度表达,促进瘤生长和免疫逃避. 向B7-H3为AML提供了一个有前途的治疗策略,同时作为预后生物标志物和治疗目标.

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Common myeloid progenitors (CMPs) are oligopotent cells that can differentiate into granulocytes and macrophages. Granulocytes and macrophages are essential for protecting the body against bacterial, viral, or fungal infections. They migrate from the bone marrow into the circulating blood to reach specific tissue sites where they differentiate and help in immune surveillance. However, they survive only for a few days and must be continuously made available to the organism to maintain a robust...
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