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Updated: Jun 28, 2025

Solubilization and Bio-conjugation of Quantum Dots and Bacterial Toxicity Assays by Growth Curve and Plate Count
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量子点和影响因素的氧化应激诱导的神经毒性

Qing Fang1, Meng Tang1

  • 1Key Laboratory of Environmental Medicine & Engineering, Ministry of Education, School of Public Health, Southeast University, Nanjing, 210009, People's Republic of China.

Nanomedicine (London, England)
|April 12, 2024
PubMed
概括

量子点 (QD) 可以治疗中枢神经系统 (CNS) 疾病,但可能导致神经毒性. 本综述详细介绍了QDs如何诱导中枢神经系统的氧化应激,影响毒性的因素以及缓解策略.

关键词:
血脑屏障 血脑屏障 血脑屏障 血脑屏障这是一个机制机制.神经毒性的作用.氧化应激是一种氧化应激.量子点是一个量子点.有活性氧物种的反应性氧物种.

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The mitochondrial electron transport chain (ETC) is the main energy generation system in the eukaryotic cells. However, mitochondria also produce cytotoxic reactive oxygen species (ROS) due to the large electron flow during oxidative phosphorylation. While Complex I is one of the primary sources of superoxide radicals, ROS production by Complex II is uncommon and may only be observed in cancer cells with mutated complexes.
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