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尿路BTEX代谢物与学龄儿童的阅读障碍几率之间的关联

Rundong Liu¹, Yanjian Wan², Bing Zhu³

¹Department of Maternal and Child Health and MOE (Ministry of Education) Key Lab of Environment and Health, School of Public Health, Tongji Medical College, Huazhong University of Science and Technology, No 13 Hangkong Road, Wuhan, 430030, Hubei, China.

Environmental science and pollution research international

|April 17, 2024

概括

暴露于代谢物,特别是酸 (MU),与儿童出现阅读障碍的几率更高有关. 这种关联在低收入家庭中更强,这表明环境因素会影响阅读发展.

科学领域:

环境健康环境健康
神经发育障碍神经发育障碍
毒理学毒理学毒理学

背景情况:

,,乙和 (BTEX) 是常见的环境污染物,具有已知的神经毒性作用.
阅读障碍是一种影响语言处理的神经发育障碍,其与BTEX暴露的联系以前尚不清楚.

研究的目的:

调查BTEX代谢物暴露与学龄儿童阅读障碍的几率之间的关联.
为了确定与增加的阅读障碍风险相关的特定BTEX代谢物.

主要方法:

一项涉及355名阅读障碍儿童和来自中国三个城市的390名对照儿童的病例对照研究.
在尿样中测量六种BTEX代谢物.
后勤回归分析,以确定代谢物水平与阅读障碍几率之间的关联.

主要成果:

尿液中含有的酸 (MU),一种的代谢物,与失阅率的增加有显著的关联 (OR=1.23).
尿路MU度的高三分位数与显著增加的阅读障碍的几率有关.
穆和阅读障碍之间的关联在来自低收入家庭的儿童中更为明显.
对于二烯,乙二烯或二烯代谢产物,没有发现显著的关联.

结论:

尿中MU度升高可能与儿童患阅读障碍的风险更高有关.
减少与MU相关的环境污染物的暴露是至关重要的,特别是对于低收入家庭的儿童.
环境因素,特别是暴露,在了解阅读障碍病因方面需要考虑.

关键词:

烯是一种烯.案例控制研究研究孩子孩子孩子孩子阅读障碍症阅读障碍症是指阅读障碍.泌尿系统 MU MU 的情况.

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