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素A4模拟物BML-111可以减少血小板激活,并保护血栓形成

Shatha AlOmar1,2, Joanne L Mitchell3, Eman AlZahrani4

  • 1Department of Clinical Laboratory Sciences, King Saud University, Prince Turki Ibn Abdulaziz Al Awwal Rd, 12371, Riyadh, Saudi Arabia. Salomar1@ksu.edu.sa.

Thrombosis journal
|April 23, 2024
PubMed
概括

甲基受体2 (FPR2/ALX) 连接体BML-111抑制了血小板激活和血栓形成. 这项研究揭示了BML-111的存在.

关键词:
在BML-111中使用.甲基受体 2/ALXX 甲基受体血小板是血小板的组成部分.血栓形成的原因之一是血栓形成.

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