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Updated: Jun 27, 2025

Preparation of Oligomeric β-amyloid1-42 and Induction of Synaptic Plasticity Impairment on Hippocampal Slices
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证据表明阿尔茨海默氏症是竞争性突触可塑性的疾病已经失败了

Zhen Huang1

  • 1Departments of Neuroscience and Neurology, University of Wisconsin-Madison, Madison, WI, USA.

Journal of Alzheimer's disease : JAD
|April 26, 2024
PubMed
概括

粉样蛋白-β (Aβ) 调节大脑突触竞争,与阿尔茨海默病 (AD) 相关的干扰. 质 ApoE 和 Aβ 寡合体相互作用以管理突触消除,睡眠在这种可塑性中起着至关重要的作用.

关键词:
阿尔茨海默氏症是阿尔茨海默氏症的一种疾病.在 ApoEEE 中.修复DNA损伤的修复粉样化物-β-β 的存在.树突性脊柱 树突性脊柱 树突性脊柱恒温的可塑性 恒温的可塑性mGluR5R5 在线观看酸盐酸氨酸的含量是多少睡眠 睡眠 睡眠 睡眠 睡眠突触性竞争 突触性竞争是指突触性竞争.

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