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微质SIX2通过上调FXYD2表达来抑制脂聚糖 (LPS) 诱导的神经炎症

Xia-Yin Cao1, Yi Liu1, Jia-Shuo Kan1

  • 1Department of Neurobiology and Cellular biology, Xuzhou Medical University, Xuzhou, Jiangsu 221004, China.

Brain research bulletin
|April 30, 2024
PubMed
概括

在炎症期间,Sine oculis homeobox同源2 (SIX2) 在微质中被重新激活,作为内源性抗炎因子. SIX2可以缓解神经炎症并保护多巴胺能神经元,为帕金森症提供新的治疗途径.

关键词:
在FXYD2上.微质细胞中的微质细胞神经炎症是一种神经炎症.帕金森病是帕金森氏症的一种疾病.第六百零二章 六十二

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The transcription factor NF-κB was discovered in 1986 in the lab of Nobel laureate Professor David Baltimore, for its interaction with the immunoglobulin light chain enhancer in B-cells. After more than three decades of study, it is now evident that NF-κB regulates the expression of over 100 genes. Most of these genes play an essential role in the innate and adaptive immune responses as well as the inflammatory responses of animals.
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