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Updated: Jun 27, 2025

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在NMD和TRP53控制原生细胞维护和大脑大小之间的表观相互作用

Lin Lin1, Jingrong Zhao1, Naoto Kubota1

  • 1Division of Biomedical Sciences, School of Medicine, University of California, Riverside, Riverside, CA 92521, USA; Center for RNA Biology and Medicine, University of California, Riverside, Riverside, CA 92521, USA.

Neuron
|May 2, 2024
PubMed
概括

无意中介的mRNA衰变 (NMD) 对大脑发育至关重要. 在小鼠中,NMD因子Upf2的缺失通过改变原生细胞周期导致小头症,通过向Trp53.3来挽救的缺陷.

关键词:
欧洲司法委员会EJC在PAX6中使用PAX6.这是TBR2的TBR2.在Upf1上.在UFF3a上.在Upf3bb上使用.细胞分裂是细胞分裂的过程.神经发生神经发生.在p2121中,在 p53 里,p53 是一个 p53 号码.原生细胞的能力.

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