该AHR抑制剂限制了抗微生物基因的表达,但不限制肠道乙氨基酸细胞中AHR依赖基因的表达

Heike Weighardt1, Michael Shapiro2, Michelle Mayer1

  • 1Immunology and Environment, Life and Medical Sciences Institute, University of Bonn, Carl-Troll-Straße 31, 53115 Bonn, Germany.

概括

阿里碳水化合物受体抑制剂 (AHRR) 不会抑制肠道乙氨基基细胞中的阿里碳水化合物受体 (AHR) 活性. 相反,AHRR限制了先天免疫和抗微生物基因表达,保持了抗炎状态.

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