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Updated: Jun 27, 2025

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吸烟诱导的CCNA2表达通过促进AT2/AT2类细胞分化来促进肺腺癌瘤发生

Qiangqiang He1, Meiyu Qu1, Chengyun Xu2

  • 1Department of Pharmacology, Zhejiang University School of Medicine, Hangzhou 310058, China.

Cancer letters
|May 4, 2024
PubMed
概括

吸烟驱动肺腺癌 (LUAD) 通过引起CCNA2过度表达,这使WNT/β-catenin信号失活并促进癌症干细胞分化.

关键词:
在AT2单元中,AT2单元是AT2单元.在CCNA2中使用CCNA2.肺部腺癌瘤是肺部腺癌.吸烟 吸烟 吸烟 吸烟在WNT/β-catenin中使用.

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