从社会压力和孤立到自主神经系统失调的自杀行为
Adrián Alacreu-Crespo1,2, Emma Sebti3,4, Rosa María Moret5
1Department of Psychology and Sociology, University of Zaragoza, C/Atarazana 4, Aragon, Teruel, 44003, Spain. aalacreu@unizar.es.
Current psychiatry reports
|May 8, 2024
概括
自主神经系统 (ANS) 的不平衡,以较高的交感神经系统 (SNS) 和较低的副交感神经系统 (PNS) 活动为标志,与自杀行为有关. 创伤和孤立等社会因素加剧了这种不平衡,影响了性.
科学领域:
- 神经科学是一个神经科学.
- 精神病学是一个精神病学.
- 自主神经系统研究 自主神经系统研究
背景情况:
- 自杀行为是复杂的,受到生物和环境因素的影响.
- 自主神经系统 (ANS) 失调在精神疾病中越来越被认可.
- 需要进一步阐明社会环境和自杀性谱系行为中的ANS活动之间的相互作用.
研究的目的:
- 审查自主神经系统 (ANS) 活动与自杀性频谱行为中的社会环境之间的关系.
- 探索社会因素是如何调解或调节ANS与自杀相关的.
- 为了确定潜在的自杀风险的ANS生物标志物.
主要方法:
- 现有文献的叙述性审查.
- 对检查同情神经系统 (SNS) 和副同情神经系统 (PNS) 活动的研究进行分析.
- 包括对儿童创伤,生活压力,孤独,孤立和社会支持等社会因素的研究.
主要成果:
- 与对照人群相比,有自杀念头或尝试的人表现出更高的SNS和较低的PNS活动.
- 脑神经系统过度活化与完成的自杀和暴力企图有关.
- 童年创伤,压力生活事件,孤独和孤立与SNS/PNS失衡相关,而社会支持可能会增强PNS控制.
结论:
- SNS / PNS 失衡可能会调解社会因素和自杀行为之间的关系.
- 自主功能障碍可以作为自杀风险的生物标志物.
- 社会背景对自杀风险较高的人群的ANS调节和弹性有重大影响.
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