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伊塔科纳酸通过化GPx4中风后神经元来保护铁性神经元

Chao Wei1, Zhongnan Xiao2, Yanling Zhang3,4

  • 1Department of Neurobiology, School of Basic Medical Sciences, Capital Medical University, Beijing, 100069, China.

Cell death and differentiation
|May 8, 2024
PubMed
概括

在脑出血后,阿康酸脱碳酶1 (Irg1) 和伊塔科纳酸保护神经元免受铁亡. 这种Irg1/itaconate-glutathione peroxidase 4 (GPx4) 途径为神经系统缺陷提供了潜在的治疗策略.

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