急性缺氧暴露对神经肌肉和血液动力学反应的影响,在步骤强度动态恒定外部阻力腿部伸展炼期间
Cierra B Ugale1, Owen F Salmon1, Matt D Segovia1
1Robbins College of Health and Human Sciences, Human & Environmental Physiology Laboratory, Baylor University, One Bear Place #97313, Waco, TX 76798, USA.
概括
急性缺氧对血液动力学反应的影响很小,但对未疲劳的个体在动态腿部延伸过程中不会影响神经肌肉反应. 这项研究探讨了在不同氧气水平下肌肉氧化和电活动.
科学领域:
- 运动生理学 运动生理学
- 运动科学 运动科学 运动科学
- 人体生理学 人体生理学
背景情况:
- 对于运动员和临床医生来说,了解对缺氧的生理反应至关重要.
- 在动态肌肉活动期间急性缺氧对神经肌肉和血液动力学效应的联合研究有限.
研究的目的:
- 调查急性诺莫西克和缺氧暴露对动态腿部延伸过程中神经肌肉和血液动力学反应的影响.
- 在不同的氧气条件下比较肌肉电活动和血液氧化.
主要方法:
- 13名休活动的男性在不同强度 (20-100%的最大强度) 进行了单边动态腿部延伸.
- 测量包括电肌图 (EMG) 幅度和频率,氧化血红蛋白 (OxyHb),脱氧化血红蛋白 (DeoxyHb),总血红蛋白 (TotalHb) 和巨大的横向组织的组织氧化 (StO2).
- 参与者在随机排序中暴露于诺莫克西克 (21% FiO2) 和低氧 (13% FiO2) 条件.
主要成果:
- 在正常和缺氧条件之间没有观察到EMG振幅或频率的显著差异.
- 随着强度的增加,EMG振幅增加,而EMG频率保持稳定,直到100%的努力.
- 虽然氧化和总血红蛋白的模式相似,但在缺氧的情况下,组织氧化 (StO2) 降低,脱氧化血红蛋白 (DeoxyHb) 增加.
结论:
- 在没有疲劳的状态下,急性低氧症在动态肌肉动作期间不会改变局部神经肌肉反应.
- 血液动力学反应,特别是肌肉氧化,受到急性低氧化的影响很小.
- 研究结果表明,神经肌肉功能在健康,娱乐活跃的男性中对急性缺氧条件具有弹性.
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