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4-Octyl itaconate通过激活KEAP1-NRF2通路在小鼠中缓解硫酸诱导的性结肠炎

Yujin Wang1, Xue Zhao1, Yifei Gao1

  • 1Department of Physiology and Pathophysiology, School of Basic Medical Sciences, Cheeloo College of Medicine, Shandong University, Wenhuaxi Road 44#Shandong Province, Jinan, China.

Inflammopharmacology
|May 20, 2024
PubMed
概括

在性结肠炎 (UC) 的小鼠模型中,4-Octyl itaconate (OI) 降低了氧化应激和结肠炎症. 氧化增强了肠道屏障功能,可能是炎症性肠病 (IBD) 的潜在治疗方法.

关键词:
4 - 八基伊塔科纳酸 4-Octyl 伊塔科纳酸肠道屏障 肠道屏障 肠道屏障核因子E2相关因子氧化应激是一种氧化应激.性结肠炎是一种

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Ulcerative colitis is a chronic inflammatory condition primarily affecting the colon and rectum. The primary drugs used in the treatment of ulcerative colitis are aminosalicylates. They exhibit anti-inflammatory and immunosuppressive properties. They modulate inflammatory mediators and inhibit the activity of nuclear factor κB (NF-κB). Aminosalicylates also reduce inflammation by inhibiting prostaglandin and leukotriene production and decreasing neutrophil chemotaxis and superoxide...
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