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Updated: Jun 25, 2025

Th17 Inflammation Model of Oropharyngeal Candidiasis in Immunodeficient Mice
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补剂受体3依赖的参与,由白的β-葡萄糖调节树突细胞,诱导调节性T细胞扩张

Areerat Kunanopparat1,2, Truc Thi Huong Dinh1,3,4, Pranpariya Ponpakdee5

  • 1Department of Microbiology, Faculty of Dentistry, Center of Excellence in Integrative Immuno-Microbial Biochemistry and Bioresponsive Nanomaterials, Chulalongkorn University , Bangkok, Thailand.

Open biology
|May 28, 2024
PubMed
概括

坎迪达格拉布拉塔菌感染会增加调节性T细胞 (Treg),抑制免疫力. 这项研究揭示了Candida glabrata对树突细胞上的补充受体3 (CR3) 的β-葡萄糖结合是Treg诱导的关键,提供了新的治疗点.

关键词:
在Candida glabrata的研究中.补充受体 3 补充受体 3树突细胞的树突细胞.免疫调节的免疫调节监管T细胞的T细胞是什么

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