布拉迪基宁2受体在实验性创伤性脑损伤后调解长期的神经认知缺陷

Antonia Clarissa Wehn1,2,3, Igor Khalin1,2,4, Senbin Hu1,2

  • 1Institute for Stroke and Dementia Research, LMU University Hospital, LMU Munich, Munich, Germany.

PubMed
概括

向布拉迪基宁2受体 (B2R) 可能减少创伤性脑损伤 (TBI) 后的慢性认知缺陷. 在TBI小鼠中,B2R缺乏保护免受长期海马体损伤和改善记忆.

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