在饮食诱导的肥胖和纤维化中,烯碳化合物受体及其抑制剂的不同细胞类型特异性功能

Frederike J Graelmann1, Fabian Gondorf1, Yasmin Majlesain1

  • 1Immunology and Environment, Life and Medical Sciences (LIMES) Institute, University of Bonn, Germany.

PubMed
概括

缺乏阿里碳化合物受体抑制剂 (AhRR) 的小鼠对饮食诱导的肥胖和代谢综合征表现出抵抗力. 骨髓细胞中增强的AhR信号保护肝脏免受损伤,而这些细胞中的AhR缺乏是有害的.

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