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Updated: Jun 23, 2025

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心脏纤维细胞增强MMP2活动,以抑制心肌细胞中缺口结合功能

Xue Bai1, Xiang Huang1, Jing Yi1

  • 1School of Anesthesiology, Guizhou Medical University, Guiyang, 550004, Guizhou, China.

Applied biochemistry and biotechnology
|June 15, 2024
PubMed
概括

重灌损伤可以导致心律失常. 低氧/低氧化治疗的心脏纤维细胞介质通过MMP2激活损害了间隙结,这表明MMP2抑制是反律乱的治疗点.

关键词:
心脏纤维细胞细胞.有条件的介质 条件的介质差距 差距的交叉点 差距的交叉点这就是MMP2的MMP2.再输血失常心律不整的情况.

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