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Updated: Jun 23, 2025

Simultaneous Measurements of Intracellular Calcium and Membrane Potential in Freshly Isolated and Intact Mouse Cerebral Endothelium
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甲基胺通过 ангиотензин II 和中间导电性激活通道诱导内皮功能障碍

Xuehui Fan1, Guoqiang Yang2, Zhen Yang3

  • 1Department of Cardiology, Angiology, Hemostaseology and Medical Intensive Care, Medical Faculty Mannheim, University Medical Centre Mannheim (UMM), Heidelberg University, Mannheim, Germany; Key Laboratory of Medical Electrophysiology of the Ministry of Education, Medical Electrophysiological Key Laboratory of Sichuan Province, Institute of Cardiovascular Research, Southwest Medical University, Luzhou, Sichuan, China; European Center for AngioScience (ECAS) and German Center for Cardiovascular Research (DZHK) partner site Heidelberg/Mannheim, Mannheim, Germany; Department of Cardiology, The Affiliated Hospital of Southwest Medical University, Luzhou 646000, China.

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
|June 18, 2024
PubMed
概括

在Takotsubo综合征中,高含量的甲醇胺因增加血管素II (Ang II) 而导致内皮功能障碍. 安格II通过ROS和PKA抑制SK4通道,损害细胞功能.

关键词:
ангиотензин II 的使用.内皮质功能障碍 内皮质功能障碍上腺素的使用情况.这就是ROSOS ROS.在SK4频道上播放.

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