CaMKIV使-1/CC2D1A5-HT1A

Kimberly Galaraga1, Anastasia Rogaeva1, Nathan Biniam1

  • 1Ottawa Hospital Research Institute (Neuroscience), Ottawa Brain and Mind Research Institute, 451 Smyth Road, Ottawa, ON K1H-8M5, Canada.

概括

类卡尔莫杜林依赖蛋白激酶 (CaMK) 酸化抑制了弗洛伊德-1基因,增加了血清素-1A受体的表达. 这种机制调节了认知发育和抗抑郁药反应.

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