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Updated: Aug 8, 2026

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats
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Published on: November 20, 2015

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由于新生儿间歇性缺氧后内部神经元不成熟导致的局部皮质功能连接异常

Ivan Goussakov, Sylvia Synowiec, Rafael Bandeira Fabres

    bioRxiv : the preprint server for biology
    |June 19, 2024
    PubMed
    概括

    在小鼠中,新生儿间歇性缺氧导致过度活跃和运动学习障碍. 这与大脑发育的改变有关,包括激发和抑制电路的变化,可通过功能性MRI检测.

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