优化心力衰竭途径以提高患者护理:优化心力衰竭患者途径计划 (PRO-HF)
Ashish Patwala1, Diane Barker1, Antoine Da Costa2
1University Hospitals of North Midlands NHS Trust, Stoke-on-Trent, UK.
ESC heart failure
|June 26, 2024
概括
优化心力衰竭患者途径计划 (PRO-HF) 帮助欧洲医院识别和解决心力衰竭护理方面的差距. 初步调查结果显示,在专家护理,数据管理和多学科团队中存在常见问题,而量身定制的干预措施显示出有前途.
科学领域:
- 心脏病学 心脏病学
- 医疗保健管理的管理
- 公共卫生 公共卫生
背景情况:
- 欧洲的医疗保健提供者面临着实施综合,多学科的心力衰竭 (HF) 护理途径的挑战.
- 欧洲心脏病学会推了针对HF管理的具体指导方针,但这些指导方针并未得到一致采用.
- PRO-HF计划是为了帮助临床医生评估和改进他们的HF患者途径而开发的.
研究的目的:
- 评估10家欧洲医院的高频路径的现状.
- 识别HF患者护理的优点,缺口和改进领域.
- 支持为路径优化制定量身定制的干预措施.
主要方法:
- 基线评估涉及使用全面的检查清单对高频服务进行整体评估.
- 数据收集包括HF护理团队的数字调查和一对一的访谈.
- 评估涵盖了四个主要领域:门诊,远程管理,设备植入/住院途径和网络最大化.
主要成果:
- 在各医院中确定了改善的关键领域,包括高频率专家护理,数据采集和分析,机构护理协议,全医院战略和多学科团队 (MDTs).
- 在所有医院中,都注意到紧急住院患者的专业护理不足.
- 缺乏明确的协议和MDT人员不足导致了护理的变化,并阻碍了采用远程患者监控等新技术.
结论:
- PRO-HF计划有效地确定了欧洲医院HF途径中的重大差距和缺陷.
- 实施建议变革的医院的初步结果令人鼓舞.
- 需要进行长期跟踪,以充分确定PRO-HF对HF途径优化和患者结果的影响.
相关概念视频
Pathophysiology of Heart Failure
1.5K
Heart failure (HF) is a progressive syndrome involving ventricles that leads to inadequate cardiac output. It can be classified based on location and output or ejection fraction. Ejection fraction (EF) is an essential measurement in the diagnosis and surveillance of HF. Reduced EF corresponds to systolic heart failure (HFrEF). However, HF with preserved ejection fraction (HFpEF) is becoming increasingly prevalent. Also known as diastolic HF, this form of HF is related to aging. The...
1.5K
Heart Failure Drugs: Inhibitors of Renin-Angiotensin System
419
The activation of the sympathetic nervous system and the renin-angiotensin-aldosterone system (RAAS) contributes to cardiac remodeling, and inhibiting the RAAS is a pharmacological target in heart failure management. As a result, neurohumoral modulation is a crucial treatment principle for managing heart failure. This approach involves using medications like ACE inhibitors (ACEIs), angiotensin receptor blockers (ARBs), β-blockers, mineralocorticoid receptor antagonists (MRAs), and neutral...
419
Heart Failure Drugs: Diuretics
362
Heart failure and kidney perfusion are interconnected in a complex way. Reduced renal perfusion and venous congestion are two significant factors that contribute to renal dysfunction in heart failure. The kidneys, primarily responsible for fluid balance in the body, are adversely affected due to compromised cardiac output and increased venous pressure. In response to reduced renal perfusion, the kidneys activate neurohumoral mechanisms to restore balance. However, these mechanisms can be...
362
Heart Failure Drugs: Inotropic Agents
564
Positive inotropic agents are commonly used as the first line of treatment for heart failure. One such agent is digoxin, derived from the genus Digitalis, which has been known for centuries but effectively utilized since 1785. However, these cardiac glycosides can have potentially toxic effects due to their mechanism of action, which involves inhibiting Na+/K+-ATPase and increasing contractility. Digoxin is absorbed orally and distributed in various tissues, including the CNS. It has a long...
564
Heart Failure Drugs: β-Blockers
336
β-adrenergic antagonists, commonly known as β-blockers, block the effects of sympathetic neurotransmitters such as noradrenaline (NA) and adrenaline (ADR). They have several beneficial effects in heart failure treatment. They reduce heart rate, the force of contraction, and cardiac muscle relaxation. They also slow the atrial-ventricular conduction rate and raise the threshold for arrhythmias. The concentration of β-blockers determines their effects on bronchodilation,...
336
Pathophysiology of Cardiac Performance
627
Typical heart performance is influenced by heart rate, rhythm, myocardial contraction, and metabolism or blood flow. The cardiac muscle exhibits distinct electrophysiological features, including pacemaker activity and calcium channel control, which play a vital role in the heart's response to various drugs. The autonomic nervous system, comprising the sympathetic and parasympathetic branches, regulates heart rate. Sympathetic activation increases heart rate, while parasympathetic activation...
627


