通过稳定CAMK2A,circADAMTS6通过驱动M2巨细胞两极化,参与吸烟引起的肺气

Jiaheng Lin1, Haibo Xia2, Jinyan Yu3

  • 1Center for Global Health, The Key Laboratory of Modern Toxicology, Ministry of Education, School of Public Health, Suzhou Institute for Advanced Study of Public Health, Gusu School, Nanjing Medical University, Nanjing 211166, Jiangsu, People's Republic of China.

PubMed
概括

香烟烟雾通过通过circADAMTS6.6增加M2巨细胞引起肺气. 这种循环RNA驱动炎症和肺损伤,为慢性阻塞性肺病 (COPD) 提供了潜在的生物标志物.