CCR2单细胞/巨细胞驱动与类固醇激素失衡相关的前列腺纤维化

Petra Popovics1,2,3,4, Samara V Silver5,6, Kristen S Uchtmann7,8

  • 1Department of Microbiology and Molecular Cell Biology, Eastern Virginia Medical School, Norfolk, VA, 23507, USA. PopoviP@evms.edu.

Scientific reports
|July 8, 2024
PubMed
概括

通过Ccr2缺乏减少单细胞透改善了良性前列腺增生 (BPH) 的小鼠模型中的尿路功能障碍和前列腺纤维化. 这突显了单细胞在BPH相关炎症和原积累中的作用.

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