FOXC1和FOXC2的切除导致异常的膜内皮细胞结合和淋巴血管形成在myxomatous中枢退化中

Can Tan1, Zhi-Dong Ge2, Shreya Kurup1,3

  • 1Department of Medicine, Feinberg Cardiovascular and Renal Research Institute (C.T., S.K., Y.D., T.L., T.K.), Feinberg School of Medicine, Northwestern University, Chicago, IL.

概括

转录因子FOXC1和FOXC2对于保持中枢 (MV) 完整性至关重要. 它们在内皮细胞中缺少导致MV缺陷和myxomatous退化,突出显示它们在心脏膜健康中的作用.