Megumi Akiyama1,2, Masashi Kanayama1, Yoshihiro Umezawa2

  • 1Department of Biodefense Research, Medical Research Institute, Tokyo Medical and Dental University (TMDU), Tokyo, Japan.

PubMed
概括

在严重炎症期间通过亡减少单细胞,可以防止组织损伤和死亡. 这种先天的免疫机制限制了促炎性细胞因子的释放,确保宿主在没有适应性免疫干预的情况下生存.

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