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Updated: Jun 19, 2025

Inducing Plasticity of Astrocytic Receptors by Manipulation of Neuronal Firing Rates
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依赖Ca2+的二酶1调节了条状脊柱投射神经元和谷氨基突触的可塑性

Shenyu Zhai1, Shintaro Otsuka1, Jian Xu1

  • 1Department of Neuroscience, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA.

Cell reports
|July 26, 2024
PubMed
概括

这项研究揭示了一种依赖氧化的长期抑郁 (NO-LTD) 在学习至关重要的脑突触. 这种可塑性机制在帕金森病模型中受损,突出显示了它在运动障碍中的作用.

关键词:
科普:神经科学是什么意思这是一种L型Ca2+) 通道.帕金森病的疾病.自闭症自闭症是什么cGMP的良好使用情况.低值尖的内部神经元.氧化氧化的使用方法基化酶是一种基化剂.有刺的投影神经元神经元.条纹体的条纹体.突触性可塑性 突触性可塑性

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