miR-93-5p通过TET3在下大脑关节出血后损害了自-解体通路

Peng-Fei Ding1, Xun-Zhi Liu2, Zheng Peng2

  • 1Department of Neurosurgery, Nanjing Drum Tower Hospital Clinical College of Nanjing Medical University, Nanjing, China; Neurosurgical Institute, Nanjing University, Nanjing, China.

Experimental neurology
|August 2, 2024
PubMed
概括

十-十一转位 (TET) 3在下大脑关节出血 (SAH) 后的神经元生存至关重要. TET3的激活改善了自-溶酶体通路 (ALP) 和神经功能,而它的下调会损害ALP并增加亡.