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Updated: Jun 18, 2025

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由HDAC2负调节的ACAP3抑制了乳头甲状腺癌细胞恶性发展

Fenfen Zhan1, Ronghui Zhang2, Lanlan Qiu2

  • 1Endocrinology department, The Second Affiliated Hospital Zhejiang University School of Medicine, China; Endocrinology department, Sanmen People's Hospital, China.

The international journal of biochemistry & cell biology
|August 4, 2024
PubMed
概括

基斯脱乙酶2 (HDAC2) 在乳头甲状腺癌 (PTC) 中负面调节 ArfGAP 具有卷轴-卷轴,膜重复和PH 域3 (ACAP3). 这种相互作用抑制了瘤的扩散和转移,同时促进了亡.

关键词:
在ACAP3中使用ACAP3.在 AKT 路径上.细胞灭亡 (apoptosis) 是一种死亡的过程.希斯脱乙酶2的使用乳头甲状腺癌是一种乳头甲状腺癌.

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