与神经精神疾病相关的神经细胞可塑性的调节由神经激素系统进行
Fei Cao1,2,3, Zhengyang Guo1,2,3, Xiaodan Ma1,2,3
1Shandong Collaborative Innovation Center for Diagnosis, Treatment and Behavioral Interventions of Mental Disorders, Institute of Mental Health, Jining Medical University, Jianshe South Road No.45, Rencheng District, Jining, Shandong, 272013, China.
Heliyon
|August 7, 2024
概括
甲基素是来自侧向下丘脑的神经,影响大脑的可塑性. 这篇评论探讨了它们在神经精神疾病 (如成,抑郁症和精神分裂症) 中的作用,提供了新的见解.
科学领域:
- 神经科学是一个神经科学.
- 神经内分泌学神经内分泌学
- 精神病学是一个精神病学.
背景情况:
- 素是侧向下丘脑产生的神经,通过特定的受体起作用.
- 氧化系统与各种生理功能有关,并与神经精神疾病有关.
- 神经可塑性在神经精神疾病中至关重要,素调节神经元可塑性.
研究的目的:
- 审查最近关于神经元可塑性中神经激素系统作用的研究.
- 探索氧系统在成,抑郁和精神分裂症中的参与.
- 为了解神经精神疾病发病过程中神经激素系统的机制提供见解.
主要方法:
- 最近科学出版物的文献综述.
- 对素,神经可塑性和神经精神疾病的研究结果的综合.
- 对,抑郁和精神分裂症的奥雷克辛能系统贡献的分析.
主要成果:
- 素在神经元可塑性中起着调节作用.
- 素系统与神经精神疾病有显著的关联.
- 有证据表明,矿石素与成,抑郁症和精神分裂症的病理生理学有关.
结论:
- 素系统是神经元可塑性的关键参与者.
- 神经激素系统的调节失调可能会导致神经精神疾病.
- 准氧化系统可能为这些疾病提供新的治疗策略.
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