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宿主-微生物相互作用在C. elegans模型中的新陈代谢重新连接,该模型是白氨酸分解缺陷的模型

Yong-Uk Lee1, Bennett W Fox2, Rui Guo1,3

  • 1Department of Systems Biology, University of Massachusetts Chan Medical School, Worcester, MA, USA.

Nature metabolism
|August 8, 2024
PubMed
概括

溶氨酸分解酶mccc-1的突变引发了C. elegans.中的代谢重新连接. 这种宿主-微生物相互作用会排毒有毒的氨酸代谢产物,并合成必要的化合物,帮助生存.

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