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Updated: Jun 17, 2025

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同类氨酸通过PGE2/STAT3信号通路促进大肠炎小鼠的肠道炎症

Akang Shao1,2, Qiu Zhao1,2, Min Chen3,4

  • 1Department of Gastroenterology, Zhongnan Hospital of Wuhan University, Donghu Road 169, Wuhan, 430071, People's Republic of China.

Digestive diseases and sciences
|August 14, 2024
PubMed
概括

超homocysteinemia恶化结肠炎通过促进T辅助17 (Th17) 细胞通过前列腺素E2 (PGE2) 和信号转换器和转录3 (STAT3) 途径的激活器分化. 这项研究阐明了结肠炎中Th17细胞增殖背后的机制.

关键词:
同类半氨酸同类半氨酸.炎症性肠病是一种炎症性肠病.在PGE2的基础上,PGE2在STAT3中,我们可以使用STAT3.17 T 辅助细胞 17

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