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Updated: May 12, 2026

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损坏的微质糖解促进了通过HK2-依赖的线粒体功能障碍在脑内出血后的炎症反应

Yin Li1, Hang Zhou1, Xuchao He1

  • 1Department of Neurosurgery & Key Laboratory of Precise Treatment and Clinical Translational Research of Neurological Diseases, Second Affiliated Hospital, School of Medicine, Zhejiang University, Hangzhou, China.

Journal of advanced research
|August 14, 2024
PubMed
概括

脑内出血 (ICH) 通过降低GLUT1和HK2的调节,损害了微质葡萄糖代谢,特别是糖解. 这种代谢转变加剧了ICH后的炎症和神经损伤.

关键词:
葡萄糖溶解是什么? 葡萄糖溶解六甲基酶 2 基酶 2炎症 炎症是一种炎症.脑内出血 脑内出血微质细胞中的微质细胞线粒体中的线粒体.

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