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Updated: May 27, 2026

Detection of Microregional Hypoxia in Mouse Cerebral Cortex by Two-photon Imaging of Endogenous NADH Fluorescence
12:03

Detection of Microregional Hypoxia in Mouse Cerebral Cortex by Two-photon Imaging of Endogenous NADH Fluorescence

Published on: February 21, 2012

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通过调节sirt2以增加细胞内NADPH,NMN部分挽救了cuproptosis

Yingying Zhang1,2, Shuting Qiu1,2, Shihan Shao1,2

  • 1School of Public Health, Hangzhou Normal University, Hangzhou, China.

Scientific reports
|August 21, 2024
PubMed
概括

尼古丁胺胺单核酸 (NMN) 通过增加保护性NADPH水平并保持铁硫蛋白质,部分地拯救细胞免受铜诱导的细胞死亡 (亡). 这发生在通过素2 (SIRT2) 的上调,独立于其胺酶活性.

关键词:
这种疾病叫做cuproptosis.对DNA损伤的反应反应它们是NADPH,NADPH.尼古丁胺胺单核酸是什么这是一个sirt2sirt2

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