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Updated: Jun 15, 2025

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通过Akt信号通路的代谢重编程,MTFR2加速肝细胞癌的发生

Zhongming Bao1, Ming Yang1, Yunhu Guo1

  • 1Department of Hepatobiliary Surgery, Huai'an Hospital Affiliated to Yangzhou University (The Fifth People's Hospital of Huai'an), Huaiyin 223300, Jiangsu, PR China.

Cellular signalling
|August 25, 2024
PubMed
概括

代谢重编程驱动肝细胞癌 (HCC). 核转录因子Y亚单元 (NFYC) 激活MTFR2,通过Akt信号传递促进HCC生长和转移. 针对这个轴提供了一个潜在的HCC疗法.

关键词:
这就是Akt信号.肝细胞癌是肝细胞癌.这就是MTFR2.代谢重编程是一种代谢重编程.美国国家财政年度委员会 (NFYC)

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