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在小鼠中,西利比宁通过TLR4/MAPK/NF-κB通路减轻引起的肺损伤

Hobin Im1, Eungyung Kim1, Hong Ju Kwon2

  • 1Department of Animal Science and Biotechnology, Research Institute for Innovative Animal Science, Kyungpook National University, Sangju-si, Republic of Korea.

In vivo (Athens, Greece)
|August 26, 2024
PubMed
概括

在暴露于氧化 (V2O5) 的小鼠中,西利比宁有效降低了炎症并改善了肺损伤. 这项研究强调了silibinininin.

关键词:
通过MAPK路径.在NF-B路径上.在NLRP3炎症酶组中,这里是Silibinin和Silibinin.它具有抗炎活性,具有抗炎活性.有助于炎症的细胞因子.肺炎是一种肺炎.氧化 (V2O5) 是一种氧化物.

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NF-κB-dependent Signaling Pathway02:26

NF-κB-dependent Signaling Pathway

The transcription factor NF-κB was discovered in 1986 in the lab of Nobel laureate Professor David Baltimore, for its interaction with the immunoglobulin light chain enhancer in B-cells. After more than three decades of study, it is now evident that NF-κB regulates the expression of over 100 genes. Most of these genes play an essential role in the innate and adaptive immune responses as well as the inflammatory responses of animals.
NF-κB-dependent Signaling Mechanism
The heterodimer of NF-κB...