肥胖改变了老年人心力衰竭风险和葡萄糖代谢障碍之间的关联:基于社区的前性队列研究
Liming Hou1,2, Xin Wang3, Peilin Li1
1Department of Cardiology, The First Affiliated Hospital of Shandong First Medical University, No. 16766, Jingshi Road, Jinan, 250014, Shandong, China.
肥胖,特别是通过腰部和部的比例来衡量的中心肥胖,降低了老年人患有葡萄糖代谢障碍的心力衰竭 (HF) 的风险. 诸如胰岛素抵抗等代谢因素在这种保护作用中起着关键作用.
科学领域:
- 老年学是一门学科.
- 心脏病学 心脏病学
- 内分泌学 在内分泌学.
背景情况:
- 葡萄糖代谢障碍是心力衰竭 (HF) 的已知危险因素.
- 肥胖症在老年人中很常见,与异常的葡萄糖代谢有关.
- 脂肪,葡萄糖代谢和HF风险之间的相互作用仍然不清楚.
研究的目的:
- 研究肥胖对老年人葡萄糖代谢障碍和HF风险之间的关联的影响.
- 探索潜在的机制,包括与脂肪相关的代谢活动.
主要方法:
- 一项对13,251名年龄≥60岁的参与者进行的队列研究.
- 基于葡萄糖代谢的分类:高血糖,糖尿病前期,不受控制的糖尿病,得到良好控制的糖尿病.
- 使用BMI,WHR和VFA评估脂肪性.
- 使用ALR,HOMA-IR和TyG对代谢活动的评估.
主要成果:
- 与高血糖相比,在糖尿病前期和糖尿病组中观察到更高的HF发病率.
- 脂肪测量 (BMI,VFA,WHR) 显著减轻了这些增加的HF风险.
- 腰部至部比 (WHR) 显示,对于HF风险,与葡萄糖代谢障碍有显著的相互作用,对于中心肥胖的可归因风险百分比为30.3%至54.4%.
- 肥胖相关的代谢因素 (ALR,HOMA-IR,TyG) 调解了肥胖和HF发病率之间的关系.
结论:
- 在老年人中,肥胖减弱了葡萄糖代谢障碍和发生的HF之间的联系.
- 腰比 (WHR) 是一个潜在的有价值的指标,用于评估肥胖在这个人群.
- 与脂肪相关的代谢活动似乎调解了脂肪对HF的保护作用,在患有葡萄糖代谢障碍的人群中.
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