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在急性病毒性心肌炎期间,GPR15介导的T细胞招募促进了病毒消除和改善结果

Bastian Stoffers1,2, Hanna Wolf1,2, Lucas Bacmeister1

  • 1Department of Cardiology and Angiology, University Heart Center Freiburg-Bad Krozingen, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Nature cardiovascular research
|August 28, 2024
PubMed
概括

缺乏GPR15会通过延迟T细胞的招募来影响病毒性心肌炎的清除,从而导致长时间的炎症和心脏功能障碍. 这突出了GPR15的重点.

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