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尼古丁胺抑制树突细胞的过度活化,通过PARP依赖信号控制自身免疫性疾病

Ai-Ping Cao1,2, Yun-Ying Wang2, Yan-Yan Shen2

  • 1Institute of Translational Medicine, Zhejiang University School of Medicine, Hangzhou 310016, China.

Nutrients
|August 29, 2024
PubMed
概括

尼古丁胺 (NAM) 有效地抑制过度活跃的树突细胞 (DC),缓解牛皮的症状. 针对NAMPT-PARP途径为DC相关的自身免疫疾病提供了一种新的治疗策略.

关键词:
在 NF-κBB 中.在PARP中,PARP是PARP.树突细胞的树突细胞.尼古丁胺胺是什么 尼古丁胺胺是什么牛皮是一种牛皮.

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