在GP VI中介的血小板激活和前凝剂活动加剧了腹腔大动脉动脉瘤中的炎症和大动脉壁重塑

Tobias Feige1, Agnes Bosbach1, Kim J Krott1

  • 1Department of Vascular and Endovascular Surgery (T.F., A.B., K.J.K., J.M., J.O., E.K., I.K., N.S., W.I., M.U.W., H.S., M.E.), University Hospital Duesseldorf, Heinrich-Heine University, Germany.

概括

血小板糖蛋白VI (GP VI) 缺乏,通过减少炎症和改善大动脉壁重塑,防止腹腔大动脉动脉瘤 (AAA) 的发展和进展. 向GP VI可能为AAA患者提供一种新的抗血小板疗法.

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