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Updated: Jun 14, 2025

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通过MOF介导的PRDX1乙化调节炎性巨细胞激活

Hui-Ru Chen1, Yidan Sun2, Gerhard Mittler2

  • 1Max Planck Institute of Immunobiology and Epigenetics, Freiburg, Baden-Württemberg, Germany; Albert-Ludwigs-University Freiburg, Faculty of Biology, Freiburg, Baden-Württemberg, Germany.

Cell reports
|August 29, 2024
PubMed
概括

MOF对氧化1 (PRDX1) 的乙化调节了巨细胞的炎症反应. 在LPS刺激期间降低PRDX1乙化,可以增强促炎信号和媒介生成.

关键词:
CP: 免疫学 免疫学科普:分子生物学 分子生物学在ERK1/2的基础上.这就是MAPKK的意思.糖溶性代谢 糖溶性代谢组织素 H3 血清素 28 酸化氨酸乙转移酶MOF是一种氨酸转移酶.巨细胞激活活动过氧化素是什么 过氧化素蛋白质乙化是蛋白质的乙化.氧化还原信号传递.信号传导的信号传导.

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