在患有中风后硬膝盖步态的患者中,肌肉协同结构发生变化
Kazuki Fujita1, Yuichi Tsushima2, Koji Hayashi3
1Graduate School of Health Science, Fukui Health Science University, 55-13-1 Egami, Fukui, 910-3190, Japan. k.fujita@fukui-hsu.ac.jp.
Scientific reports
|August 31, 2024
概括
脑卒中后膝盖硬是由于简化肌肉协同作用而产生的,而不是单个肌肉问题. 了解这些改变的肌肉协调模式是有效康复策略的关键.
科学领域:
- 神经科学是一个神经科学.
- 生物力学 生物力学
- 康复科学 康复科学 康复科学
背景情况:
- 脑卒中后的膝盖硬 (SKG) 与异常的肌肉活动有关,但肌肉之间的相互作用仍然不太清楚.
- 识别潜在的肌肉协调模式对于开发有针对性的干预措施至关重要.
研究的目的:
- 为了研究和表征肌肉协同模式,在行走的个人中,在中风后的SKG.
- 为了比较中风患者与SKG,中风患者没有SKG和健康对照之间的肌肉协同作用.
主要方法:
- 一项涉及20名中风后SKG患者,16名非SKG中风患者和15名健康对照者的横截面研究.
- 在10米的步行过程中,从六个下肢肌肉中收集了电肌图数据.
- 非负矩阵因子化 (NMF) 用于分析肌肉协同作用,推导出时间变化的活性 (C),肌肉重量 (W) 和变异占比 (tVAF).
主要成果:
- 与对照组相比,SKG集团的占总差异 (tVAF) 百分比较高,这表明协同效应复杂性发生了变化.
- 特定的肌肉协同模块显示出异常的激活模式:初始姿势模块 (膝盖延伸器) 在摆动过程中过度活跃,初始摆动模块 (部曲器,脚骨曲器) 在姿势和摆动阶段显示出改变的活动.
- 在SKG组的整体肌肉协同结构似乎简化了,在协同激活中出现了明显的异常.
结论:
- 脑卒中后的膝盖硬与简化肌肉协同结构和涉及多个肌肉的异常激活模式有关.
- 针对个体肌肉控制的康复方法可能不如针对改变的肌肉协同网络的疗效.
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