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Updated: Jul 20, 2026

Ferritinophagy: Assessing the Selective Degradation of Iron by Autophagy in Human Fibroblasts
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葡萄糖依赖性胰岛素型多 (GIP) 通过Epac/Rap1信号通路缓解老化诱导的大脑损伤中的铁亡

Jiwon Ko1, Soyoung Jang2, Soyeon Jang1

  • 1Institute of Life Science and Biotechnology, Kyungpook National University, Daegu 41566, Korea.

BMB reports
|September 2, 2024
PubMed
概括

葡萄糖依赖型胰岛素型多 (GIP) 保护神经元免受铁亡,这是一种与衰老和神经退行性疾病相关的细胞死亡过程. 这种激素通过Epac/Rap1通路保持神经元的完整性,提供治疗潜力.

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Dipeptidyl Peptidase 4 Inhibitors01:23

Dipeptidyl Peptidase 4 Inhibitors

Dipeptidyl peptidase 4 (DPP-4) is a serine protease widely distributed in the body. It's involved in the inactivation of GLP-1 and GIP hormones, which are crucial for insulin regulation. DPP-4 inhibitors, such as sitagliptin (Januvia), saxagliptin (Onglyza), linagliptin (Tradjenta), alogliptin (Nesina), and vildagliptin (Galvus), help increase the proportion of active GLP-1, enhancing insulin secretion. These inhibitors work by competitively binding to DPP-4. This binding causes a significant...