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Updated: Jun 14, 2025

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细颗粒物通过铁灭症在实验性自身免疫性脑膜炎中增强了Th17细胞的致病性

Yaning Liu1, Wanyun Zhang1, Hongmiao Wang1

  • 1The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.

Ecotoxicology and environmental safety
|September 4, 2024
PubMed
概括

细颗粒物 (PM2.5) 暴露会通过促进细胞死亡途径铁亡,使实验性自身免疫性脑膜炎 (EAU) 恶化. 这突显了铁化作为PM2.5诱导的脑膜炎的潜在治疗标.

关键词:
这是自身免疫性疾病.实验性的自身免疫性脑膜炎.铁性化 (ferroptosis) 是一种炎症 炎症是一种炎症.对于PM2.5来说,这是一个很好的例子.

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When T cells with CD4 markers are activated, they give rise to two types of effector cells: helper T cells and regulatory T cells. Meanwhile, T cells with CD8 markers differentiate into effector cytotoxic T cells. The differentiation of CD4 T cells into helper T cell subsets, such as Th1, Th2, and Th17 cells, is dependent on the antigen type, antigen-presenting cell, and regulatory cytokines.
Th1 cells stimulate dendritic cells to express necessary co-stimulatory molecules on their surfaces for...
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