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CCL25有助于D-Gal/LPS诱导的急性肝衰竭的发生

Fei Sun1, Jingwei Wang1, Xiangfen Ji1

  • 1Department of Hepatology, Qilu Hospital (Qingdao) of Shandong University, Qingdao, China.

Journal of gastroenterology and hepatology
|September 5, 2024
PubMed
概括

通过NF-κB信号传导促进巨细胞炎症,CCL25 (CCC 化学因子连接体 25) 加剧急性肝衰竭 (ALF). 抑制CCL25显示了ALF的治疗潜力.

关键词:
CCL25 CCL25 CCL25 CCL25 CCL25 CCL25 CCL25 CCL25在 NF-κB 信号传输中.急性肝衰竭是一种严重的肝衰竭.巨细胞是什么?巨细胞是什么?

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