2008年至2023年胃潰瘍的新興趨勢和熱點:一篇經典評量分析
Jiahui Li1, Jiamei Jin2, Xiaoyang Wang2
1Haiyan Hospital of Traditional Chinese Medicine, Jiaxing, China.
Medicine
|September 10, 2024
概括
本文献分析揭示了2008-2023年胃病 (PU) 的研究趋势. 中国在出版物方面处于领先地位,其中"氧化应激"作为一个关键的研究热点,指导未来的胃潰瘍研究.
科学领域:
- 胃肠病学和消化系统疾病
- 圣经计量学和科学计量学
- 医学研究 分析 分析
背景情况:
- 胃潰瘍 (PU) 是一種普遍存在的胃腸關節疾病.
- 图书统计分析在医学研究中越来越多地被使用.
- 以前没有报道过关于胃潰瘍研究的综合文献计量研究.
研究的目的:
- 为了进行胃潰瘍 (PU) 研究的文献分析.
- 在过去15年 (2008-2023) 中确定PU研究的趋势和热点.
- 为未来在该领域进行深入研究和开发提供基础.
主要方法:
- 分析了来自Web of Science核心集合 (2008-2023) 的文献数据.
- 使用了包括CiteSpace,VOSviewer和SCImago Graphica在内的可视化工具.
- 总共有6,491篇论文被收集并根据定义的标准进行分析.
主要成果:
- 中国发表的论文最多;贝勒医学院是领先的机构.
- 亚马奥卡·约西奥是最多产的作者,而马尔费尔泰纳·彼得获得了最多的引用.
- 世界胃肠病学杂志发表了大多数论文;胃肠病学是被引用最多的期刊. "氧化应激"成为一个热门的关键词.
结论:
- 本文献分析提供了关于胃的研究趋势和热点的全面概述.
- 这些发现有助于快速了解当前的胃病研究格局.
- 这项研究对于旨在推进胃潰瘍研究的研究人员来说是一个有价值的参考资料.
相关概念视频
01:30Peptic Ulcer Disease I: Introduction
144
Peptic Ulcer Disease (PUD) is characterized by mucosal excavation in the esophagus, stomach, pylorus, or duodenum. It can manifest as acute or chronic based on the extent and duration of mucosal involvement.
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
An acute ulcer, marked by superficial erosion and minimal inflammation, swiftly resolves upon identifying and addressing the underlying cause. In contrast, a chronic ulcer persists, potentially eroding through the muscular wall and forming fibrous tissue.
Peptic ulcers can also be...
144
01:26Peptic Ulcer Disease IV: Management
75
Medical treatment strategies for peptic ulcers encompass various methods. The primary goal of treatment is to diminish gastric acidity and strengthen mucosal defense mechanisms.
The therapeutic approach involves ensuring adequate rest, implementing drug therapy, promoting smoking cessation, making dietary modifications, and emphasizing long-term follow-up care.
Pharmacological management
The prevailing therapy for peptic ulcers involves a combination of managing the patient's current...
The therapeutic approach involves ensuring adequate rest, implementing drug therapy, promoting smoking cessation, making dietary modifications, and emphasizing long-term follow-up care.
Pharmacological management
The prevailing therapy for peptic ulcers involves a combination of managing the patient's current...
75
01:28Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies
97
Peptic ulcer disease (PUD) presents with diverse symptoms depending on the location and severity of the ulcer. Clinical manifestations of peptic ulcer include dull pain and a burning sensation in the mid-epigastric region.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
97
01:20Drugs for Peptic Ulcer Disease: Prostaglandin Analogs as Mucosal Protective Agents
375
The gastric mucosa produces prostaglandins E2 (PGE2) and prostacyclin (PGI2), crucial in maintaining gastric health. They exert cytoprotective effects, including increasing bicarbonate secretion, releasing protective mucin, reducing gastric acid output, and preventing harmful vasoconstriction. These effects are mediated through various receptors, such as EP1, EP2, EP3, and EP4.
Non-steroidal anti-inflammatory drugs (NSAIDs) can induce peptic ulcers by inhibiting cyclooxygenase, decreasing...
Non-steroidal anti-inflammatory drugs (NSAIDs) can induce peptic ulcers by inhibiting cyclooxygenase, decreasing...
375
01:24Pathophysiology of Peptic Ulcer Disease: Mucosal Defense Factors
358
Peptic ulcer disease, commonly called PUD, represents a multifaceted condition characterized by disruptions in the lining of the gastrointestinal (GI) tract. Central to the protection of the gastrointestinal lining is the mucosal-bicarbonate barrier. This physiological defense mechanism is a formidable shield against the corrosive effects of gastric acid and pepsin secretion in the stomach. Its role is pivotal in maintaining the structural integrity of the stomach's inner lining.
358
01:28Peptic Ulcer Disease II: Pathophysiology
322
Peptic Ulcer Disease (PUD) is characterized by the development of ulcers in the stomach or duodenal mucosa. Its pathophysiology is complex, involving a balance between damaging and protective elements.
Damaging agents such as Helicobacter pylori, gastric acid, pepsin, and nonsteroidal anti-inflammatory drugs (NSAIDs) can weaken the mucosal defense, allowing hydrogen ions to infiltrate back and harm epithelial cells.
Damaging agents such as Helicobacter pylori, gastric acid, pepsin, and nonsteroidal anti-inflammatory drugs (NSAIDs) can weaken the mucosal defense, allowing hydrogen ions to infiltrate back and harm epithelial cells.
322