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Updated: May 13, 2026

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卡伦杜洛西德E 通过Sirtuin 2-NLRP3 炎症酶轴改善脂肪组织中的炎症反应

Aini Yuan1, Jing Liu1, Jianan Guo1

  • 1School of Life Sciences, Zhejiang Chinese Medical University, Hangzhou 310053, China.

Journal of agricultural and food chemistry
|September 16, 2024
PubMed
概括

糖化合物Calenduloside E (CE) 通过抑制NLRP3炎症酶激活来对抗与肥胖相关的炎症. 它通过增强Sirtuin 2 (SIRT2) 活性来实现这一目标,为代谢障碍提供了潜在的治疗方法.

关键词:
在NLRP3中,NLRP3是NLRP3中的一个.在SIRT2中,SIRT2是SIRT2.脂肪组织的脂肪组织.卡尔度化物E E巨细胞是什么?巨细胞是什么?热灭症是什么?

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NF-κB-dependent Signaling Pathway02:26

NF-κB-dependent Signaling Pathway

The transcription factor NF-κB was discovered in 1986 in the lab of Nobel laureate Professor David Baltimore, for its interaction with the immunoglobulin light chain enhancer in B-cells. After more than three decades of study, it is now evident that NF-κB regulates the expression of over 100 genes. Most of these genes play an essential role in the innate and adaptive immune responses as well as the inflammatory responses of animals.
NF-κB-dependent Signaling Mechanism
The heterodimer of NF-κB...