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Updated: Jun 13, 2025

Induction of Intestinal Inflammation by Adoptive Transfer of CBir1 TCR Transgenic CD4+ T Cells to Immunodeficient Mice
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Induction of Intestinal Inflammation by Adoptive Transfer of CBir1 TCR Transgenic CD4+ T Cells to Immunodeficient Mice

Published on: December 16, 2021

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益生菌胰岛素通过调节肠道微生物群和短链脂肪酸来控制Th17细胞介导的中枢神经系统自身免疫力

Ning Li1,2,3, Xinyan Han1, Ming Ruan2,3

  • 1The MOE Key Laboratory for Standardization of Chinese Medicines and the MOE Innovation Centre for Basic Medicine Research on Qi-Blood TCM Theories, Shanghai Key Laboratory of Compound Chinese Medicines, Institute of Chinese Materia Medica, Shanghai University of Traditional Chinese Medicine, Shanghai, China.

Gut microbes
|September 17, 2024
PubMed
概括

益生菌胰岛素通过调节肠道细菌和减少炎症Th17细胞,降低了小鼠多发性硬化症 (MS) 的严重程度. 这表明胰岛素是MS患者潜在的治疗药物.

关键词:
在这种情况下,胰岛素和胰岛素是不同的.在Th17细胞中.我们的肠道微生物群.这是一种炎症性脱髓化.多发性硬化症 多发性硬化症短链脂肪酸 短链脂肪酸

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