""/apo/holo SOD1:ALS

Faezeh Ashkaran1, Bagher Seyedalipour2, Payam Baziyar1

  • 1Department of Molecular and Cell Biology, Faculty of Basic Science, University of Mazandaran, Babolsar, Iran.

BMC chemistry
|September 19, 2024
PubMed
概括

超氧化物脱酶 (SOD1) 中的突变和金属缺乏导致蛋白质错误折叠和聚合,导致粉样蛋白的形成. 这有助于神经退行性疾病,如ALS.

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